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Conclusion AST emerges as a promising therapeutic option in countering the oxidative stress and inflammatory responses triggered by FPN exposure in the rat brain

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[27] A gene expressed in glial cells actively transports glutamate into the extracellular space, [27] while, in the nucleus accumbens-stimulating group II metabotropic glutamate receptors, this gene was found to reduce extracellular glutamate levels

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Their complementary mechanisms make the combination suitable for users focusing on recovery and structural performance as part of a broader regimen