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10.1371/journal.pone.0216828 11 ChenL.JoshiP.PiatkivskyiA.AguilarK.LinJ

Postoperative glucocorticoid withdrawal syndrome is usually characterized by biochemical evidence of HPAA suppression, with many signs and symptoms consistent with cortisol deficiency despite the use of supraphysiologic doses of glucocorticoids

GLP-1R activity also promotes the transactivation of epidermal growth factor receptor (EGFR), which then signals through phosphoinositide 3-kinase (PI3K) and insulin receptor substrate-2 (IRS-2), and subsequently activates extracellular-signal-regulated kinase 1 and 2 (ERK1/2) and nuclear translocation of PKC to mediate -cell proliferation and differentiation (110, 114)

We explored these limitations with meta-regression, as well as with subgroup analysis, and the results were unchanged